PS370 Unit 6 Tobacco and Substance Use Paper Example

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How a composite scallop captain recovered from opioid use disorder but still smokes at sea is the subject of this PS370 Unit 6 tobacco and substance use paper. His opioid use began with a 2009 back injury and oxycodone prescribed so he could keep fishing, a pattern a Massachusetts report found among fishing and construction workers. Volkow's brain disease model and Benowitz's account of nicotine show both dependencies running through the same reward circuits and conditioned cues. Cochrane reviews support buprenorphine and combined smoking treatment, and the paper argues his tobacco use deserves the medical treatment his opioid use received. Purdue Global PS 370, Health Psychology, assigns this paper in the BS in Psychology.

CoursePS370 Health Psychology
UnitUnit 6
Paper typeTobacco and substance use paper
LengthAbout 1,120 words, 7 pages double-spaced
FormatAPA 7 student paper
SchoolPurdue University Global
ProgramBS in Psychology
UpdatedOctober 2026

Free sample paper for PS370 Unit 6

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Two Dependencies, One Recovery: A PS370 Unit 6 Tobacco and Substance Use Paper

[Student Name]

Purdue University Global

PS370: Health Psychology

Unit 6 Tobacco and Substance Use Paper

[Instructor Name]

[Date]

The captain, his history and his treatment are composite; the report, reviews and studies cited are real.

What this part is doingComparing two dependencies in one person lets the paper ask why one was treated as a disease and the other as a habit.
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A Fishing Port and an Injury

Paul, now 51, has fished out of New Bedford, Massachusetts, since he left high school. His opioid use began the way it did for many people in his industry. In 2009 he herniated a disc in his lower back while working a dredge in rough weather. A doctor prescribed oxycodone, and for several years the pills let him keep fishing through pain. Commercial fishing has among the highest injury rates of any job, and crew members who do not fish are not paid.

Massachusetts later documented the pattern. A state analysis of opioid-related overdose deaths from 2011 to 2015 by industry and occupation found that construction and extraction workers had the highest death rate and farming, fishing and forestry workers the next highest, both far above the average for all workers (Massachusetts Department of Public Health, 2018). The report linked these rates to high injury rates and to jobs without paid sick leave or job security, which push people to work through pain.

By 2015 Paul's prescriptions had been cut back, and he was buying pills on the street. When those became too expensive, he spent about a year injecting heroin. A friend's overdose on the dock in 2017 led him to a treatment program, where he started buprenorphine. Seven years on, he has stayed off illicit opioids. Tobacco is the older dependency: about twenty cigarettes a day since age seventeen, and he still smokes at sea fourteen months after a heart attack.

Two Dependencies in the Brain

Volkow et al. (2016) summarized the brain disease model as a repeating loop with three phases. In binge and intoxication, a drug produces large surges of dopamine in reward circuits, and cues associated with the drug come to trigger dopamine release on their own. In withdrawal and negative affect, stress systems become more active and the reward system less responsive, so that the person uses partly to escape feeling bad. In preoccupation and anticipation, the prefrontal regions that normally put the brakes on impulses work less well, so cravings win more often. They argued that these changes explain why people keep using despite serious harm and why relapse risk lasts long after use stops.

Paul's opioid use and his smoking run through the same reward circuits, but only one of them was ever treated as a medical condition.

Nicotine follows a similar path. Benowitz (2010) described how nicotine acts on nicotinic receptors in the brain, releasing dopamine and other neurotransmitters, and how repeated exposure leads to tolerance and a withdrawal syndrome of irritability, poor concentration, anxiety and craving. He emphasized that smoking is maintained not only by the drug but by conditioned cues, the settings, times and moods in which a smoker has used thousands of times.

Paul's night watches are a powerful set of such cues. For thirty years he has lit a cigarette in the wheelhouse after the first tow, during the steam to the next bed and before dawn. On land those cues are absent, and his success at quitting there fits Benowitz's account. On the boat, every cue is in place, and the craving returns on the second night of every trip.

The brain disease model has critics. Some argue that it understates choice and social conditions and can make people feel powerless. Paul's case shows why both views matter: his opioid use began with an injury and a prescription, was shaped by an industry that does not pay sick days, and ended with a medical treatment that changed his brain's response to opioids.

What this part is doingIncluding the critique and then showing how the case fits both sides avoids treating a contested model as settled.
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What Treatment Research Shows

For opioid use disorder, the evidence for buprenorphine is strong. A Cochrane review found that people on buprenorphine maintenance stayed in treatment longer than those on placebo, and that medium and high doses reduced illicit opioid use; it performed similarly to methadone at adequate doses, though methadone retained slightly more people at flexible doses (Mattick et al., 2014). For Paul, buprenorphine had a practical advantage: he could take it daily on a boat, without traveling to a clinic, once his prescriber agreed to a supply for each trip.

For smoking, the evidence favors combining medication with behavioral support. A Cochrane review of 53 trials found that combined pharmacotherapy and behavioral support increased quit rates by roughly 70 to 100 percent compared with brief advice or usual care (Stead et al., 2016). Paul has never received either. His cardiologist told him to quit, and his recovery program, focused on opioids, did not address tobacco.

That gap is common, and Paul noticed it himself. His recovery counselor once asked about his drinking and his sleep at every visit but never about cigarettes, and when Paul raised it, the counselor said to focus on one thing at a time. Seven years later, the one thing has been handled, and the other has helped put a stent in his heart. Smoking rates among people in treatment for opioid use disorder are far higher than in the general population, and smoking contributes heavily to their deaths. Treating tobacco as a lesser problem in recovery reflects a belief that quitting smoking might threaten sobriety, but studies of smoking cessation in addiction treatment have generally not found that quitting increases relapse.

A Plan for the Remaining Dependency

Paul's opioid recovery provides a model for his tobacco treatment. It worked because he received a medication that addressed his brain's dependence, regular contact with a counselor and a plan for high-risk situations. His smoking at sea needs the same three elements.

First, medication: a nicotine patch combined with a short-acting form such as lozenges for the night watch, or varenicline, which his cardiologist would need to approve. Second, support between trips through a quitline or his rehabilitation program, using the same relapse-prevention skills he learned in opioid treatment. Third, a cue plan for the boat: no cigarettes on board, a different routine for the first tow and the steam, and a nonsmoking wheelhouse. His counselor could point out that he has already done this once, with a harder drug.

What this part is doingBuilding the smoking plan from what worked in his opioid recovery uses the patient's own success as the evidence he will trust most.
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Conclusion

Paul's two dependencies share a biology and a history of being shaped by work. His opioid use was treated as a medical condition, with an effective medication and support, and he recovered. His smoking has been treated as a habit he should simply drop. The research supports treating both the same way, and his heart makes the second one urgent.

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References

Benowitz, N. L. (2010). Nicotine addiction. New England Journal of Medicine, 362(24), 2295-2303. https://doi.org/10.1056/NEJMra0809890

Massachusetts Department of Public Health. (2018). Opioid-related overdose deaths in Massachusetts by industry and occupation, 2011-2015.

Mattick, R. P., Breen, C., Kimber, J., & Davoli, M. (2014). Buprenorphine maintenance versus placebo or methadone maintenance for opioid dependence. Cochrane Database of Systematic Reviews, 2014(2), Article CD002207. https://doi.org/10.1002/14651858.CD002207.pub4

Stead, L. F., Koilpillai, P., Fanshawe, T. R., & Lancaster, T. (2016). Combined pharmacotherapy and behavioural interventions for smoking cessation. Cochrane Database of Systematic Reviews, 2016(3), Article CD008286. https://doi.org/10.1002/14651858.CD008286.pub3

Volkow, N. D., Koob, G. F., & McLellan, A. T. (2016). Neurobiologic advances from the brain disease model of addiction. New England Journal of Medicine, 374(4), 363-371. https://doi.org/10.1056/NEJMra1511480

What the PS370 Unit 6 assignment asks for

In Unit 6, PS370 asks you to examine tobacco and other substance use from a health psychology perspective. Describe the person's use and its history, including social and occupational factors. Explain dependence using current research on the brain and on learning, and note at least one critique of the model you use. Summarize the health effects relevant to the person. Evaluate the evidence for treatment, citing reviews where possible, and recommend a plan that fits the person's history. Write about substance use in nonjudgmental, person-first language. Use APA style with scholarly sources and government reports where relevant, in a paper of about five pages. Composite cases are expected for sensitive histories.

How the PS 370 Unit 6 example is put together

The paper opens with Paul's injury, prescriptions, street pills, a year of injecting heroin and a friend's overdose that led him to buprenorphine. A section on the port cites the Massachusetts analysis of overdose deaths by occupation. A neuroscience section explains the three-stage addiction cycle and nicotine's conditioned cues, under a highlighted line that only one dependency was ever treated as medical, and notes critiques of the brain disease model. A treatment section summarizes Cochrane findings on buprenorphine and combined smoking treatment, with the counselor who never asked about cigarettes. A plan section rebuilds his smoking treatment from the three elements that worked for opioids. The conclusion argues that his heart now makes the second dependency urgent.

PS370 Unit 6 rubric: what earns full points

Markers of the Unit 6 paper want dependence explained accurately, in a way that connects biology to the person's behavior, such as cues tied to a setting. Social and occupational causes should be included with sources, not assumed. A critique of the brain disease model shows balance. Treatment evidence earns credit when it reports what reviews found and for whom. Recommendations should follow from the person's history rather than a standard list. Many rubrics also score respectful, person-first wording. References to government reports and Cochrane reviews should be complete and formatted correctly in APA style, with an honest note on what the evidence leaves uncertain.

Common PS370 Unit 6 mistakes, and how to avoid them

Substance use papers lose points when they describe use as a moral failure or use labels like addict. Another frequent weakness explains dopamine in detail but never connects it to the person's cravings or settings. Students sometimes treat smoking as harmless next to other drugs. Treatment sections often name therapies without evidence or ignore medication. Some papers omit social causes, such as work injuries or lack of sick leave. Before submitting, reread for language, link each biological claim to a behavior you described, cite at least one review for every treatment you recommend, and show how the person's history shapes the plan. Those steps protect both accuracy and tone.

Get a PS370 Unit 6 example written to your instructions

Share the substance and the instructions with the rubric. A custom paper explains dependence, health effects and evidence-based treatment. Turnaround is 24-48h, free for a first request. The paper above is an original model document written by our desk, not a submitted student paper and not an official Purdue University Global document.

More PS370 papers and BS in Psychology samples

PS370 Unit 6 questions, answered

Is addiction a brain disease?

The brain disease model is influential and debated; it describes changes in reward and control systems.

Does buprenorphine work?

Reviews find it keeps people in treatment and reduces illicit opioid use.

Why is quitting smoking so hard?

Nicotine dependence combines withdrawal, conditioned cues and habit.

Where can I find a free PS370 Unit 6 sample paper?

This page holds the complete PS370 Unit 6 paper on a composite captain's opioid recovery and continued smoking, with treatment evidence for both.

What does the PS370 Unit 6 sample recommend for smoking?

Combined medication and support, a cue plan for night watches and a smoke-free wheelhouse, built on what worked in his opioid recovery.